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Steffan Nawrocki, PhD

Co-Program Leader, Clinical and Translational Oncology Program
Professor, College of Medicine
Steffan T. Nawrocki, PhD, is a Professor in the Department of Medicine and Co-Leader of the Clinical and Translational Oncology Program (CTOP) at the University of Arizona Comprehensive Cancer Center (UACCC). He also serves as Co-Director of Translational Research, where he helps drive the center’s strategy to advance discovery science into early-phase clinical trials. 

Nawrocki is a nationally recognized leader in experimental therapeutics whose work has redefined how protein homeostasis can be exploited for therapeutic gain. He trained at The University of Texas M.D. Anderson Cancer Center and completed postdoctoral studies at St. Jude Children’s Research Hospital, establishing a foundation in mechanistic cancer biology that has translated into a sustained and impactful drug development program. His research has been instrumental in advancing novel therapeutic strategies that target fundamental survival pathways in cancer, including protein degradation, autophagy, and stress-response signaling networks. These efforts have helped establish new paradigms for overcoming drug resistance and have directly informed the clinical development of multiple investigational therapies. More recently, his work has expanded to define emerging targets such as p97/VCP, REDD1, lysosomal vulnerabilities, and oncolytic viral therapy with broad relevance across hematologic malignancies and solid tumors. 

Nawrocki leads a highly collaborative and well-funded research program supported by the National Institutes of Health and other major agencies, and he has authored a substantial body of influential publications in cancer biology and translational science. At the UACCC, Nawrocki plays a central role in shaping a high-impact translational research environment that integrates mechanistic discovery with therapeutic development. Through his leadership in CTOP, he helps align multidisciplinary teams to accelerate the progression of promising therapies into investigator-initiated clinical trials, strengthening the center’s position as a leader in experimental therapeutics. 

His contributions have been recognized by multiple honors, including the University of Arizona Cancer Center Inaugural Research Prize for Translational Science and the College of Medicine Faculty Excellence Award for Basic and Translational Investigation. Through a sustained focus on innovation, rigor, and clinical relevance, Nawrocki’s work is advancing next-generation therapeutic strategies designed to overcome resistance and improve outcomes for patients with cancer.
 
Research Program Role
Clinical and Translational Oncology Program

Display Name
Steffan Nawrocki, PhD

Publications

Carew JS, Espitia CM, Sureshkumar S, Carrera Espinoza MJ, Gamble ME, Wang W, Lee B, Nawrocki ST. 2025. REDD1 is a determinant of the sensitivity of renal cell carcinoma cells to autophagy inhibition that can be therapeutically exploited by targeting PIM activity. Cancer Letters. 613.
Nawrocki ST, Espitia C, Carrera Espinoza MJ, Jones TM, Gamble ME, Sureshkumar S, Chang M, Wang W, Carew JS. 2024. Inhibition of autophagy antagonizes breast cancer brain metastogenesis and augments the anticancer activity of lapatinib. Clinical and Translational Medicine. 14.
Nawrocki ST, olea J, Villa CC, Dadrastoussi H, Wu K, Tsao-Wei D, Colombo A, Coffey M, Fernandez HE, Chen X, et al. 2023. Comprehensive single-cell immune profiling defines the patient multiple myeloma microenvironment following oncolytic virus therapy in a Phase 1b trial. Clinical Cancer Research. 29:5087–5103.
Jones TM, Espitia CM, Chipollini J, Lee B, Wertheim JA, Carew JS, Nawrocki ST. 2023. Targeting NEDDylation is a Novel Strategy to Attenuate Cisplatin-induced Nephrotoxicity. Cancer Research Communications. 3:245–257.
Jones TM, Espitia CM, Ooi A, Bauman JE, Carew JS, Nawrocki ST. 2022. Targeted CUL4A inhibition synergizes with cisplatin to yield long-term survival in models of head and neck squamous cell carcinoma through a DDB2-mediated mechanism. Cell Death and Disease. 13.
Kelly KR, Espitia CM, Zhao W, Wu K, Visconte V, Anwer F, Calton CM, Carew JS, Nawrocki ST. 2018. Oncolytic reovirus sensitizes multiple myeloma cells to anti-PD-L1 therapy. Leukemia. 32:230–233. doi:10.1038/leu.2017.272.
Kelly KR, Espitia CM, Zhao W, Wu K, Visconte V, Anwer F, Calton CM, Carew JS, Nawrocki ST. Oncolytic reovirus sensitizes multiple myeloma cells to anti-PD-L1 therapy. Leukemia. 32:230–233.